Tinea capitis

Tinea capitis is named after a dermatophyte fungus for the infection of scalp. Although common in children, tinea capitis is seen less often in adults.

Hair trichophyton (which can be briefly infected as “T”) and microscopy (“M”.) Fungus.

In New Zealand, M. canis is the most common dermatology fungus that causes tinea capitis. This fungus is zoophilic i.e. it naturally grows on animals rather than humans. M. canis tinea capitis is an infected cat or rarely due to the contact of an old cat or dog.

Other zephylic fungus sometimes causes tinea capitis:

•  T. Verrucosum (evolution of cattle)
•  T. Mentagrophytes var Equinoxes
•  M Nainam (exit from pigs)
•  M. Distortum (a version of M Kentee found in cats)

In the United States, T. Tonuran also has become a common cause of tinea capitis; It is passed from person to person because it naturally infects humans (i.e. it is an anthropogenic). It often does not produce any symptoms and is usually found in adult carriers.

Other anthropophilic fungus sometimes causes tinea capitis:

•  Especially among African patients. Violaceum
•  M Audouinii
•  M. Ferruginium
•  T. Schoenleinii
•  T. Rubrum
•  T. Magnie
•  T. Soundness
•  T. Yondai

Dermatology fungus is sometimes produced in soil (geological organisms). It rarely causes tinea capitis:

•  M. Gpsm
•  M Fulvam

Types of Tinea Capitis Transition
Tang capitals are classified as how the fungus attacks the hair shaft.

Actactrix infection
Attack of the acetothrix hair, M. Canis, M. Audienne, M. Distortum, M. Ferruginium, M. Gypsum, M Nainam and T. Because of infection with Varrucosam is due. Fungal branches (Haifes) and spiers (arthrocronidia) come out of the hair. The ectothrhex infection can be detected by the affected area of ​​Woods Lighthouse (long wave ultraviolet light) examination, the vet uses it to examine the fur of its cats. Fahr is fluorosis when infected with M canis.

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Endotrips infection
The consequences of endothront invasion t. Tonuran, T. Vioresium and T. Are accompanied by infection with soundness. The hair shaft is full of fungal branches (Haifes) and spiers (arthrocronidia). Endotrips infection is not fluorosis with woods light.

Favus
Fews do not happen in New Zealand. It t. Schoenleinii is caused by infection, resulting in the destruction of honey of the hair shaft.

Clinical features of tinea capitis
Tinea capitis is the most popular among 3 to 7 years of age. Compared to girls, it is a bit common in boys. T. Transition through tonuran may occur in adults.

T. Anthropophilic infections such as tonuran are more common in the living conditions of the crowd. The fungus can contaminate with hairbrushes, clothes, towels and seats. Diseases survive for a long time and can later infect another individual months.

Zoophilic infections are caused by direct contact with an infected animal and usually do not go from person to person.

Geophilic infections are usually produced while working in infected soils but are sometimes transferred from the infected animal.

Tinea capitis can be present in many ways.

•  Dry scaling – like dandruff, but usually with hair loss with kites
•  Black point – Hair scalp breaks down on the surface, which is scaly
•  Smooth area of ​​hair fall
•  Carion – very swollen mass, like a boil
•  Favers – Yellow Crust and Mated Hair
•  Carrier state has no symptoms and only light scaling (T. Tununan).

The result of tinea capitis can be swollen lymph glands in the back of the neck. The untreated Kerian and Faws may result in permanent scarring (bald areas).

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It can also be an ID response, especially after starting antifangal treatment.

Tinea capitis diagnosis
If there is a combination of scale and bald patches, tinea capitis is suspected. Wood light fluorescence is helpful but not clinical because it is only positive if the responsible organism is fluorosis, and fluorescence is sometimes seen for other reasons.

Diagnosis of Tinea capitis should be confirmed by skin scraping microscopes and culture and hair pulled by roots (see laboratory test).

Occasionally, a diagnosis is made on skin biopsy, which shows the specific histopathological characteristics of tinea capitis.

Treatment of Tinea Capitis
Tinea capitis is usually used to treat oral antifungal drugs, including grisofulwyn (which is no longer available in New Zealand), terbinaafine and itraconazole.

Carrier treatment
If the child has anthropophilic infection, then all family members should be screened for signals of infection. Brushing of scalp areas of scalp should be taken for mycology. Sometimes it is best to treat the entire family whether fungal infections have been proven or not.

It is advisable for parents of classmates and other players to check their children and to be treated if necessary. In some countries, infected children are not allowed to go to school. Elsewhere, children with Tinnaean capitis can participate in school so that they are receiving treatment.

The carrier can not have any symptoms. Treatment of carriers is necessary to prevent the spread of infection. Antifangle shampoo can be twice weekly weekly for two weeks but if the cultures are positive, oral treatment is recommended.

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The appropriate shampoos include:

•  2.5% selenium sulfide
•  1% to 2% zinc pierethion
•  povidone iodine
•  2% Ketoconazole

Treatment of Tinea Capitis
Tinea capitis needs treatment with an oral antifungal agent. Griseofulvin is probably the most effective agent for infection with the microscope canis, but is no longer available in New Zealand. Scalop Trikofetan infection can be successfully eliminated from 4 to 6 weeks using oral tarbinafine, itraconazole or fluaconazole. However, these drugs are not always successful and some other agent may need to try. Temporary treatment can also be determined eg. Once weekly dose.

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